K562

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1.
Conclusion: Tricholoma Matsutake is able to inhibit the proliferation and induce the differentiation of K562 cells.
结论：松茸是抑制白血病细胞增殖并诱导其分化的天然诱导剂，但其机理有待进一步探讨。
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2.
Conclusion: The growth inhibition and apoptosis induction of K562 cells treated by DFMO were associated with suppressed telomerase activity.
结论DFMO引起的K562细胞生长抑制及凋亡诱导与端粒酶活性抑制有关。
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3.
Conclusion Etoposide chemotherapy combined with hyperthermia showed obvious inhibitive effect to K562 cells.
结论热疗联合足叶乙甙能增强对K562细胞的体外抑制作用；
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4.
The K562 cells treated with toosendanin extracts showed morphological characteristics of apoptotic cells.
处理组细胞可见典型的凋亡形态学改变；
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5.
Result: Tricholoma Matsutake markedly inhibited K562 cell proliferation.
结果：松茸对K562细胞增殖具有明显的抑制作用。
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6.
The cytotoxicity assays detected NK activity to K562 cells in patients with non secreting pituitary adenoma and healthy donor.
细胞毒性实验的方法检测垂体无分泌腺瘤患者和正常人外周血中NK细胞对K562的杀伤活性。
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7.
Objective To observe the effects of Triptolide on proliferation and apoptosis of erythroleukemia K562 in vitro.
目的探讨雷公藤内酯醇对人类红白血病K562细胞株增殖和凋亡的影响。
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8.
K562 cells under iron deprivation was also confirmed by western blot analysis.
K562细胞剥夺下铁也证实了免疫印迹分析。
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9.
MTT method to observe the effects of different concentrations of serum and Rubus parvifolius saponin on K562 cell proliferation.
MTT法观察不同浓度的含药血清和茅莓总皂苷对K562细胞株的增殖作用的影响。
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10.
AIM: To explore how Glivec and HMBA affect the cell growth, apoptosis, cell cycle and cyclin protein expression of K562 cells.
目的：研究格列卫和六亚甲基二乙酰胺（HMBA）对K562细胞增殖、凋亡、细胞周期及周期蛋白表达的影响。
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